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      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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    <loc>https://vibrantrevival.com/vibrantrevivalblog/thyroid-for-fertility</loc>
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      <image:title>Blog - Optimizing Thyroid for Fertility - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - Optimizing Thyroid for Fertility - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - Optimizing Thyroid for Fertility - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - Optimizing Thyroid for Fertility - Make it stand out</image:title>
      <image:caption>The frequency of pulsatile release of gonadotropin-releasing hormone (GnRH) from the hypothalamus is often increased in polycystic ovary syndrome (PCOS), leading to luteinizing hormone (LH) hypersecretion by the pituitary gland. LH hypersectretion + altered regulation of ovarian processes, including increased sensitivity of ovarian theca cells to LH, results in excess androgen production. Hyperinsulinemia further sensitizes ovarian theca cells to LH, leading to even more androgen production.  Androgens work with follicle-stimulating hormone (FSH) to luteinize follicles by inducing LH receptors.  In a healthy ovary, one dominant follicle is hyper-responsive to the luteinizing process and accumulates 10X more LH receptors than other follicles; allowing it to continue to grow while remaining follicles become growth-inhibited and are reabsorbed back into the body.  Conversely, in a state of androgen excess, premature luteinization of follicles impair selection of the dominant follicle, causing follicle maturation arrest, an ovulation, + polycystic ovarian morphology (PCOM). Anovulatory cycles prevent the production of progesterone.  High concentrations of progesterone are required to suppress the increased LH pulse frequency of PCOS. Low to no progesterone further promotes excess androgen production. Sex hormone-binding globulin (SHBG) is suppressed by androgens, insulin resistance + hypothyroidism, leading to increased levels of free + active testosterone. Notably, genetic, epigenetic, and environmental factors contribute considerably to the expression of these alterations.  Image borrowed with permission from Springer Nature. Nat Rev Dis Primers 2016;2:16057. Polycystic ovary syndrome, Azziz R, Carmina E, Chen Z, Dunaif A, Laven JS, Legro RS, et al. Copyright 2016.Azziz. Polycystic Ovary Syndrome. Obstet Gynecol 2018. All Content is the intellectual property of Vibrant Revival.</image:caption>
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      <image:title>Blog - Functional Fertility Care&amp;nbsp;&amp;nbsp; - Make it stand out</image:title>
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      <image:title>Blog - Functional Fertility Care&amp;nbsp;&amp;nbsp; - Make it stand out</image:title>
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      <image:title>Blog - Functional Fertility Care&amp;nbsp;&amp;nbsp; - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - Functional Fertility Care&amp;nbsp;&amp;nbsp; - Make it stand out</image:title>
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      <image:title>Blog - Functional Fertility Care&amp;nbsp;&amp;nbsp; - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - Functional Fertility Care&amp;nbsp;&amp;nbsp; - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - Functional Fertility Care&amp;nbsp;&amp;nbsp; - Make it stand out</image:title>
      <image:caption>The frequency of pulsatile release of gonadotropin-releasing hormone (GnRH) from the hypothalamus is often increased in polycystic ovary syndrome (PCOS), leading to luteinizing hormone (LH) hypersecretion by the pituitary gland. LH hypersectretion + altered regulation of ovarian processes, including increased sensitivity of ovarian theca cells to LH, results in excess androgen production. Hyperinsulinemia further sensitizes ovarian theca cells to LH, leading to even more androgen production.  Androgens work with follicle-stimulating hormone (FSH) to luteinize follicles by inducing LH receptors.  In a healthy ovary, one dominant follicle is hyper-responsive to the luteinizing process and accumulates 10X more LH receptors than other follicles; allowing it to continue to grow while remaining follicles become growth-inhibited and are reabsorbed back into the body.  Conversely, in a state of androgen excess, premature luteinization of follicles impair selection of the dominant follicle, causing follicle maturation arrest, an ovulation, + polycystic ovarian morphology (PCOM). Anovulatory cycles prevent the production of progesterone.  High concentrations of progesterone are required to suppress the increased LH pulse frequency of PCOS. Low to no progesterone further promotes excess androgen production. Sex hormone-binding globulin (SHBG) is suppressed by androgens, insulin resistance + hypothyroidism, leading to increased levels of free + active testosterone. Notably, genetic, epigenetic, and environmental factors contribute considerably to the expression of these alterations.  Image borrowed with permission from Springer Nature. Nat Rev Dis Primers 2016;2:16057. Polycystic ovary syndrome, Azziz R, Carmina E, Chen Z, Dunaif A, Laven JS, Legro RS, et al. Copyright 2016.Azziz. Polycystic Ovary Syndrome. Obstet Gynecol 2018. All Content is the intellectual property of Vibrant Revival.</image:caption>
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      <image:title>Blog - PCOS Pathophysiology: Types + Phenotypes - Make it stand out</image:title>
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      <image:title>Blog - PCOS Pathophysiology: Types + Phenotypes - Make it stand out</image:title>
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      <image:title>Blog - PCOS Pathophysiology: Types + Phenotypes - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - PCOS Pathophysiology: Types + Phenotypes - Make it stand out</image:title>
      <image:caption>The frequency of pulsatile release of gonadotropin-releasing hormone (GnRH) from the hypothalamus is often increased in polycystic ovary syndrome (PCOS), leading to luteinizing hormone (LH) hypersecretion by the pituitary gland. LH hypersectretion + altered regulation of ovarian processes, including increased sensitivity of ovarian theca cells to LH, results in excess androgen production. Hyperinsulinemia further sensitizes ovarian theca cells to LH, leading to even more androgen production.  Androgens work with follicle-stimulating hormone (FSH) to luteinize follicles by inducing LH receptors.  In a healthy ovary, one dominant follicle is hyper-responsive to the luteinizing process and accumulates 10X more LH receptors than other follicles; allowing it to continue to grow while remaining follicles become growth-inhibited and are reabsorbed back into the body.  Conversely, in a state of androgen excess, premature luteinization of follicles impair selection of the dominant follicle, causing follicle maturation arrest, an ovulation, + polycystic ovarian morphology (PCOM). Anovulatory cycles prevent the production of progesterone.  High concentrations of progesterone are required to suppress the increased LH pulse frequency of PCOS. Low to no progesterone further promotes excess androgen production. Sex hormone-binding globulin (SHBG) is suppressed by androgens, insulin resistance + hypothyroidism, leading to increased levels of free + active testosterone. Notably, genetic, epigenetic, and environmental factors contribute considerably to the expression of these alterations.  Image borrowed with permission from Springer Nature. Nat Rev Dis Primers 2016;2:16057. Polycystic ovary syndrome, Azziz R, Carmina E, Chen Z, Dunaif A, Laven JS, Legro RS, et al. Copyright 2016.Azziz. Polycystic Ovary Syndrome. Obstet Gynecol 2018. All Content is the intellectual property of Vibrant Revival.</image:caption>
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      <image:title>Blog - PCOS: Diagnosis,  Signs + Symptoms - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - PCOS: Diagnosis,  Signs + Symptoms - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - PCOS: Diagnosis,  Signs + Symptoms - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - PCOS: Diagnosis,  Signs + Symptoms - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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      <image:title>Blog - PCOS: Diagnosis,  Signs + Symptoms - Make it stand out</image:title>
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      <image:title>Blog - PCOS: Diagnosis,  Signs + Symptoms - Make it stand out</image:title>
      <image:caption>Whatever it is, the way you tell your story online can make all the difference.</image:caption>
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